Background and activities
Björn Gustafsson is Dean at the Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology (NTNU). He is Professor of Gastroenterology. Gustafsson is also specialist in Internal Medicine and Gastroenterology, and an attending physician at St. Olavs University Hospital in Trondheim.
His research background is translational research on inflammatory bowel disease, as well as basic research in neuroendocrinology and neuroendocrine tumor biology.
Björn Gustafsson was president of The Norwegian Gastroenterological Association from 2009 until 2013.
Before he became Dean he was Vice Dean (Dean of Research) at the Faculty of Medicine from 2012-2015.
Scientific, academic and artistic work
A selection of recent journal publications, artistic productions, books, including book and report excerpts. See all publications in the database
- (2020) Intestinal Epithelial Cells Express Immunomodulatory ISG15 During Active Ulcerative Colitis and Crohn's Disease. Journal of Crohn's and colitis.
- (2018) Not only stem cells, but also mature cells, particularly neuroendocrine cells, may develop into tumours: time for a paradigm shift. Therapeutic Advances in Gastroenterology. vol. 11.
- (2018) Tu1753 - type 1 Interferon Signaling in Intestinal Epithelial Cells During Active Inflammatory Bowel Disease. Gastroenterology. vol. 154 (6).
- (2016) Cellular localization of guanylin and uroguanylin mRNAs in human and rat duodenal and colonic mucosa. Cell and Tissue Research. vol. 365 (2).
- (2015) The guanylate cyclase-C signaling pathway is down-regulated in inflammatory bowel disease. Scandinavian Journal of Gastroenterology. vol. 50 (10).
- (2014) The PAS positive material in gastric cancer cells of signet ring type is not mucin. Experimental and molecular pathology (Print). vol. 96 (3).
- (2014) Mucosal toll-like receptor 3-dependent synthesis of complement factor B and systemic complement activation in inflammatory bowel disease. Inflammatory Bowel Diseases. vol. 20 (6).
- (2013) Relevance of TNBS-Colitis in Rats: A Methodological Study with Endoscopic, Historical and Transcripttomic Characterization and Correlation to IBD. PLOS ONE. vol. 8 (1).
- (2013) Su1771 Reduced Guanylate Cyclase C Signaling in IBD May Sustain Inflammation, Epithelial Barrier Dysfunction and Increase Cancer Risk in IBD. Gastroenterology. vol. 144 (5).
- (2013) The Stimulatory Adenosine Receptor ADORA2B Regulates Serotonin (5-HT) Synthesis and Release in Oxygen-Depleted EC Cells in Inflammatory Bowel Disease. PLOS ONE. vol. 8 (4).
- (2013) Whole Genome Gene Expression Meta-Analysis of Inflammatory Bowel Disease Colon Mucosa Demonstrates Lack of Major Differences between Crohn's Disease and Ulcerative Colitis. PLOS ONE. vol. 8 (2).
- (2013) REG gene expression in inflamed and healthy colon mucosa explored by in situ hybridisation. Cell and Tissue Research. vol. 352 (3).
- (2013) Gastric Carcinoids (Neuroendocrine Neoplasms). Gastroenterology Clinics of North America. vol. 42 (2).
- (2013) In situ hybridization in human and rodent tissue by the use of a new and simplified method. Applied immunohistochemistry & molecular morphology (Print). vol. 21 (2).
- (2012) Tu1912 Endoscopic, Histological and Transcriptomic Characterization of TNBS-Colitis in Rats, a Model for IBD. Gastroenterology. vol. 142.
- (2012) The role of mechanical forces and adenosine in the regulation of intestinal enterochromaffin cell serotonin secretion. American Journal of Physiology - Gastrointestinal and Liver Physiology. vol. 302 (3).
- (2012) Dissecting the IBD Transcriptome. Inflammatory Bowel Diseases. vol. 18.
- (2012) Differential signal pathway activation and 5-HT function: the role of gut enterochromaffin cells as oxygen sensors. American Journal of Physiology - Gastrointestinal and Liver Physiology. vol. 303 (10).
- (2012) Microencapsulation of small intestinal neuroendocrine neoplasm cells for tumor model studies. Cancer Science. vol. 103 (7).
- (2012) Systemic leptin administration in supraphysiological doses maintains bone mineral density and mechanical strength despite significant weight loss. Endocrinology. vol. 153 (5).